The majority of pediatric/infant cardiac arrests are due to hypoxia. airway embarrassment (food bolus, mucus, vomitus, blood, or tongue), prolonged seizure, poisoning, hypoxic atmosphere, airway trauma, etc. Hypoxic cardiac arrests: drowning, drug overdose, choking assault, electrocution, mechanical asphyxiation (plastic bag). Putting aside for the moment the new findings about giving too much oxygen: (Note reply above by BLS SYSTEMS citing actual scientific measurements.) There's a place for the tube (maybe), but it's not in routine cardiac arrest.ĮDIT: Another thought: even in those rare cases where intubation may be beneficial to a select subset of patients in cardiac arrest, is that benefit so great as to offset the harm caused by decreasing blood return to the heart secondary to increased intrathoracic pressure with ventilations? Finally, if patients in cardiac arrest are aspirating or losing their airways so frequently that intubation during cardiac arrest is as vital as you claim, then why is there no evidence of increased survival in cardiac arrest patients following intubation? And ETCO2 can be measured via a nasal cannula. Most cardiac arrests are not due to blocked airways. As for your second comment, I'll again point out that cardiac arrest due to some respiratory etiology makes up a very small percentage of all cardiac arrests.
Ewy would disagree with you that the NRB is a "temporizing measure until an ALS airway can be placed". Why should we treat all cardiac arrests as due to hypoxia when the reality is that most are due to heart disease?Īgain, I think Dr.
Cardiac arrest secondary to hypoxia is an exceedingly rare thing.
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